The RNA m6A Reader YTHDF2 Maintains Oncogene Expression ...

YTHDF2 links RNA epitranscriptomic modifications and GSC growth, laying the foundation for the YTHDF2-MYC-IGFBP3 axis as a specific and novel therapeutic ...

The RNA m6A Reader YTHDF2 Maintains Oncogene Expression and Is a Targetable Dependency in Glioblastoma Stem Cells

The RNA m6A Reader YTHDF2 Maintains Oncogene Expression ...

Glioblastoma is a universally lethal cancer driven by glioblastoma stem cells (GSC). Here, we interrogated -methyladenosine (m6A) mRNA modifications in GSCs ...

(PDF) The RNA m6A Reader YTHDF2 Maintains Oncogene ...

S1G). We next investigated whether differential localization of. m6A peaks informed GSC biology. We generated a GSC-. speciﬁc gene expression signature ...

The RNA m6A reader YTHDF2 maintains oncogene expression and ...

YTHDF2 links RNA epitranscriptomic modifications and GSC growth, laying the foundation for the YTHDF 2-MYC-IGFBP3 axis as a specific and ...

The RNA m6A reader YTHDF2 controls NK cell antitumor and ...

, et al. 2021 . The RNA m6A reader YTHDF2 maintains oncogene expression and is a targetable dependency in glioblastoma stem cells . Cancer Discov. 11. : 480.

The RNA m6A Reader YTHDF2 Maintains Oncogene Expression ...

Abstract Glioblastoma is a universally lethal cancer driven by glioblastoma stem cells (GSC). Here, we interrogated N6-methyladenosine (m6A) mRNA ...

Comprehensive analysis of m6A reader YTHDF2 prognosis ...

Dixit, et al. The RNA m6A reader YTHDF2 maintains oncogene expression and is a targetable dependency in glioblastoma stem cells. Cancer ...

Supplementary Data from The RNA m6A Reader YTHDF2 Maintains ...

Dixit et al. YTHDF2 maintains glioblastoma stem cells. The RNA m6A reader YTHDF2 maintains oncogene expression and is a targetable dependency in

The RNA m6A Reader YTHDF2 Is Essential for the Post ... - Cell Press

Results · YTHDF2 Is a Cytoplasmic Protein Expressed at All Stages of Mammalian Gametogenesis · Ythdf2 Deficiency Is Partially Permissive in Mice ...

RNA m6A reader YTHDF2 facilitates precursor miR-126 maturation ...

We found that YTHDF2 is aberrantly overexpressed in acute myeloid leukemia (AML) patients, especially in relapsed patients, and plays an oncogenic role in AML.

The role of m6A modification in the biological functions and diseases

The RNA m6A reader YTHDF2 maintains oncogene expression and is a targetable dependency in glioblastoma stem cells. Cancer Discov. https ...

Combating cancer stem cells: RNA m6A methylation and ... - Frontiers

The RNA m6A reader YTHDF2 maintains oncogene expression and is a targetable dependency in glioblastoma stem cells. Cancer Discov. 11 (2), 480–499. doi ...

shoot the reader

The rna m6a reader ythdf2 maintains · oncogene expression and is a targetable · dependency in glioblastoma stem cells. Cancer Discov. 11, 480 ...

METTL3-mediated m6A modification of LINC00839 maintains ...

The RNA m6A reader YTHDF2 maintains oncogene expression and is a targetable dependency in glioblastoma stem cells. Cancer Discov. 2021;11 ...

Role of m6A writers, erasers and readers in cancer

METTL3/YTHDF2 reduces the expression of ZBTB4 mRNA in a m6A ... The RNA m6A reader YTHDF2 maintains oncogene expression and Is a targetable ...

YTHDF2 facilitates UBXN1 mRNA decay by recognizing METTL3 ...

The RNA m6A Reader YTHDF2 maintains oncogene expression and is a targetable dependency in glioblastoma stem cells. Cancer Discov. 2021;11 ...

m6A Modifications Play Crucial Roles in Glial Cell ... - Frontiers

The RNA m6A reader YTHDF2 maintains oncogene expression and is a targetable dependency in glioblastoma stem cells. Cancer Discov (2020). doi: 10.1158/2159 ...

Research Progress on the Role of RNA m6A Modification in Glial ...

The RNA m6A Reader YTHDF2 Maintains Oncogene Expression and Is a Targetable Dependency in Glioblastoma Stem Cells. Cancer Discov. 2021, 11, 480–499. [Google ...

The mRNA m 6 A reader YTHDF2 suppresses proinflammatory ...

The m6A reader YTHDF2 is a novel regulator of inflammatory pathways in HSCs. YTHDF2 loss upregulates m6A-modified proinflammatory ...